Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Then again, our ability to rescue SERT purpose in KI midbrain synaptoneurosomes from the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by greater pFAK localization in five-HT https://virgill531ozl3.wikiinside.com/user