Preliminary reports suggest conolidine might inhibit distinct ion channels, minimizing neuronal excitability and limiting suffering alerts. This system is especially pertinent in neuropathic pain, where by irregular signaling leads to persistent pain. Additionally, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to agony notion. Computational docking e... https://davyi391avs2.signalwiki.com/user